Posts filed under 'Sugar'




Stop the Progress of Prediabetes

You can prevent this precursor to type 2 diabetes from developing into full-blown disease.
By Madeline Vann, MPH
Medically reviewed by Cynthia Haines, MD

By some estimates, one-third of adults in the United States have a condition called prediabetes; 13 percent have type 2 diabetes. Prediabetes may be more common in men (36 percent) than in women (23 percent).

Prediabetes means that while your blood sugar levels are higher than normal, that level isn’t high enough to warrant a diabetes diagnosis. However, a prediabetes diagnosis means it is time for action to prevent diabetes. “In simple terms, there is a gap between what we call diabetes, which is a fasting blood sugar of 126 and above, and normal, which is less than 100 fasting,” explains Vivian Fonseca, MD, a professor of medicine and pharmacology and chief of endocrinology at Tulane University Health Sciences Center in New Orleans.

“In between,” he continues, “you have impaired fasting glucose. If you do a glucose tolerance test, and you are in the gap, you have prediabetes. You are at risk for getting diabetes in the future and you are also at risk for heart disease.”

Type 2 Diabetes: Prevention

If are told your blood sugar is abnormally high, you’ve just had a red flag waved in front of you. You’re being warned that unless you make some changes in your life today, your future will probably include a diabetes diagnosis.

“Walking 30 minutes a day and reducing weight by 5 percent can decrease the risk [of getting type 2 diabetes] by 60 percent over three years,” says Dr. Fonseca. While there are medications that have the same effect, lifestyle change is less expensive and has fewer side effects, Fonseca says.

Cutting your weight is crucial. “One of the links with obesity is that fat induces a mild low-grade inflammation throughout the body that contributes to heart disease and diabetes,” Fonseca explains. Without making any changes, you could develop type 2 diabetes within 10 years of first developing prediabetes.

Type 2 Diabetes: Who Should be Tested?

Prediabetes is a “silent” condition, says Fonseca. While some people may experience symptoms of diabetes such as fatigue or increased urination, most people’s blood sugar rises without any outward signs at all. This means you might not know you need to be tested for prediabetes — and even if you are screened, your doctor might not give you all the information you need to prevent it.

For these reasons, diabetes experts developed criteria for those who should be tested. The American Diabetic Association recommends that any adult age 45 or older should be tested for diabetes and prediabetes.

The ADA also recommends that any adult under age 45 who is overweight and has at least one of the following risk factors should be tested:

* Family history (especially parent or sibling with diabetes)
* Physically inactive lifestyle
* Native American, African-American, or Hispanic heritage
* Prior gestational diabetes diagnosis
* Birth of a baby over nine pounds in weight
* High blood pressure or treatment for high blood pressure
* Polycystic ovarian syndrome (PCOS) diagnosis
* Dark, velvety rash around the armpits or neck
* History of heart disease

If your test reveals that you have prediabetes, you should be tested again in one to two years, depending on your doctor’s recommendations.

Type 2 Diabetes: Types of Tests

There are two tests used to screen for diabetes and prediabetes:

* Fasting plasma glucose: a test of your blood after you haven’t eaten for eight hours (usually overnight)
* Oral glucose tolerance test: a comparison of your blood taken first after eight hours without food (fasting) and then two hours later after you have consumed a sugary drink given to you by the lab technician.

If you fit the screening criteria listed above, make an appointment to get tested as soon as possible. It could be the first step toward preventing the development of type 2 diabetes.
Everyday Health Network

Add comment September 7, 2009

Eating Healthy Chocolate!

Debbie Mormino, in connection with Elaine Iagatta, Dr. Wallach, and Youngevity, are excited to announce the debut of Tru Chocolate™ (http://ChocolateFromGod.com) starting off in the US. This is the ALTERNATIVE SUPER SNACK, that will be an important addition to help people world wide with the increasing problem of WEIGHT ISSUES.
 
Chocolate lovers all over the world are asking, “What Is Tru Chocolate”?
 
Tru Chocolate™ is: 
*Good for you and good tasting!
*GUILT FREE, SUGAR FREE / HEALTHY WEIGHT LOSS CHOCOLATE!
*100% Natural, but still tastes great; it has been formulated to be compliant with the health requirements of the vegan communities
*Gluten free and Kosher
*Diabetics, hypoglycemic, and lactose intolerant individuals can all eat this chocolate with no worries or ill side effects!
*Helps with weight management/loss while promoting healthy blood sugar levels and helping to satiate your appetite
*Sweetened with ALL NATURAL Xylitol and loved by dentists for it dental health benefits
*Tru Chocolate™ kills harmful bacteria in the mouth, improving the health of teeth and gums
*Overflowing with powerful antioxidants and all natural ingredients
*Ridiculously high ORAC value (3040 per piece!) compare it to Goji or Noni per serving
*Only 36 calories per serving

Chocolate is the universal language and Tru Chocolate™ is the latest in the ALL NATURAL weight management system and whole food. It is engineered with entirely natural ingredients. In addition, it has been formulated to be compliant with the health requirements of the Vegetarian communities. This new product is focused on the main objectives to lower and control blood sugar, to increase lean body mass, and to establish a solid platform for the restoration of health.
 

  TRU Chocolate™ “WOW”  – The Razzle Dazzle After-Taste

Most people feel an amazing after-taste when they eat Tru Chocolate™. After consuming the chocolate wafer, immediately drink cold water.  “Wow,” your water just became chocolate milk!  It is wild!   This is an experiential, “must-try-to-believe” product!
 
The amazing chocolate after experience is like a sweet taste with a bit of spice added to it that stays in your palate for about 15 – 30 minutes after consuming Tru Chocolate™. This is the result of several of the ingredients in Tru Chocolate™  —  and indeed, it is an indication that it is working!
 
Tru Chocolate™ is a “Functional Super Food,” i.e. it has a nutraceutical function in addition to its nourishment value. The razzle dazzle after-taste arises from three ingredients:
 Bio Flavenoids (from the citrus extract)- are 2000 times sweeter than sugar. It is strong enough for a few molecules to stay in the palate to create that sweet after-taste
Bioperin – is a black pepper derivative enhances the perception of taste. Anything you eat or drink after Tru Chocolate™ will taste stronger
Fabenol – is a lentil derivative blocks the absorption of sugar and, therefore, increases the sensation of sweetness.
The after-taste is an advantage. It induces an individual to drink water, which is very important for weight loss and the body.  Because it makes water taste sweet, one can drink simple water, hydrate the body, and be satiated, instead of filling-up on soda or juice (which are generally high in calories, carbs and sugar).

Known as the “Guilt-Free Chocolate,” Tru Chocolate was created as a healthy and tasty treat for calorie conscious people (who still have taste buds).  Today, it is a delicious treat everyone can enjoy.
All Natural, Toxin Free Ingredients: Organic Cocoa Liqueur, Organic Cocoa Butter, Xylitol, Proprietary Herbal Formula, Natural Vanilla Extract and Organic Lecitin. A proprietary blend containing Momordica Charantia, Dehydrated Noni Fruit, Bioflavonoids (Citrus Extract), Ellagic Acid (from pomegranate), Green Tea Extract, Fabanol®** and Bioperine®** (from black pepper).Note:  These statements have not been evaluated by the FDA. This is not intended to diagnose, cure or prevent any disease.

Add comment June 25, 2009

76 Ways Sugar Can Ruin Your Health

 by Nancy Appleton, Ph.D
Author of the book Lick The Sugar Habit

In addition to throwing off the body’s homeostasis, excess sugar may result in a number of other significant consequences. The following is a listing of some of sugar’s metabolic consequences from a variety of medical journals and other scientific publications.

  1. Sugar can suppress your immune system and impair your defenses against infectious disease.1,2
  2. Sugar upsets the mineral relationships in your body: causes chromium and copper deficiencies and interferes with absorption of calcium and magnesium. 3,4,5,6
  3. Sugar can cause can cause a rapid rise of adrenaline, hyperactivity, anxiety, difficulty concentrating, and crankiness in children.7,8
  4. Sugar can produce a significant rise in total cholesterol, triglycerides and bad cholesterol and a decrease in good cholesterol.9,10,11,12
  5. Sugar causes a loss of tissue elasticity and function.13
  6. Sugar feeds cancer cells and has been connected with the development of cancer of the breast, ovaries, prostate, rectum, pancreas, biliary tract, lung, gallbladder and stomach.14,15,16,17,18,19,20
  7. Sugar can increase fasting levels of glucose and can cause reactive hypoglycemia.21,22
  8. Sugar can weaken eyesight.23
  9. Sugar can cause many problems with the gastrointestinal tract including: an acidic digestive tract, indigestion, malabsorption in patients with functional bowel disease, increased risk of Crohn’s disease, and ulcerative colitis.24,25,26,27,28
  10. Sugar can cause premature aging.29
  11. Sugar can lead to alcoholism.30
  12. Sugar can cause your saliva to become acidic, tooth decay, and periodontal disease.31,32,33
  13. Sugar contributes to obesity.34
  14. Sugar can cause autoimmune diseases such as: arthritis, asthma, multiple sclerosis.35,36,37
  15. Sugar greatly assists the uncontrolled growth of Candida Albicans (yeast infections)38
  16. Sugar can cause gallstones.39
  17. Sugar can cause appendicitis.40
  18. Sugar can cause hemorrhoids.41
  19. Sugar can cause varicose veins.42
  20. Sugar can elevate glucose and insulin responses in oral contraceptive users.43
  21. Sugar can contribute to osteoporosis.44
  22. Sugar can cause a decrease in your insulin sensitivity thereby causing an abnormally high insulin levels and eventually diabetes.45,46,47
  23. Sugar can lower your Vitamin E levels.48
  24. Sugar can increase your systolic blood pressure.49
  25. Sugar can cause drowsiness and decreased activity in children.50
  26. High sugar intake increases advanced glycation end products (AGEs)(Sugar molecules attaching to and thereby damaging proteins in the body).51
  27. Sugar can interfere with your absorption of protein.52
  28. Sugar causes food allergies.53
  29. Sugar can cause toxemia during pregnancy.54
  30. Sugar can contribute to eczema in children.55
  31. Sugar can cause atherosclerosis and cardiovascular disease.56,57
  32. Sugar can impair the structure of your DNA.58
  33. Sugar can change the structure of protein and cause a permanent alteration of the way the proteins act in your body.59,60
  34. Sugar can make your skin age by changing the structure of collagen.61
  35. Sugar can cause cataracts and nearsightedness.62,63
  36. Sugar can cause emphysema.64
  37. High sugar intake can impair the physiological homeostasis of many systems in your body.65
  38. Sugar lowers the ability of enzymes to function.66
  39. Sugar intake is higher in people with Parkinson’s disease.67
  40. Sugar can increase the size of your liver by making your liver cells divide and it can increase the amount of liver fat.68,69
  41. Sugar can increase kidney size and produce pathological changes in the kidney such as the formation of kidney stones.70,71
  42. Sugar can damage your pancreas.72
  43. Sugar can increase your body’s fluid retention.73
  44. Sugar is enemy #1 of your bowel movement.74
  45. Sugar can compromise the lining of your capillaries.75
  46. Sugar can make your tendons more brittle.76
  47. Sugar can cause headaches, including migraines.77
  48. Sugar can reduce the learning capacity, adversely affect school children’s grades and cause learning disorders.78,79
  49. Sugar can cause an increase in delta, alpha, and theta brain waves which can alter your mind’s ability to think clearly.80
  50. Sugar can cause depression.81
  51. Sugar can increase your risk of gout.82
  52. Sugar can increase your risk of Alzheimer’s disease.83
  53. Sugar can cause hormonal imbalances such as: increasing estrogen in men, exacerbating PMS, and decreasing growth hormone.84,85,86,87
  54. Sugar can lead to dizziness.88
  55. Diets high in sugar will increase free radicals and oxidative stress.89
  56. High sucrose diets of subjects with peripheral vascular disease significantly increases platelet adhesion.90
  57. High sugar consumption of pregnant adolescents can lead to substantial decrease in gestation duration and is associated with a twofold increased risk for delivering a small-for-gestational-age (SGA) infant.91,92
  58. Sugar is an addictive substance.93
  59. Sugar can be intoxicating, similar to alcohol.94
  60. Sugar given to premature babies can affect the amount of carbon dioxide they produce.95
  61. Decrease in sugar intake can increase emotional stability.96
  62. Your body changes sugar into 2 to 5 times more fat in the bloodstream than it does starch.97
  63. The rapid absorption of sugar promotes excessive food intake in obese subjects.98
  64. Sugar can worsen the symptoms of children with attention deficit hyperactivity disorder (ADHD).99
  65. Sugar adversely affects urinary electrolyte composition.100
  66. Sugar can slow down the ability of your adrenal glands to function.101
  67. Sugar has the potential of inducing abnormal metabolic processes in a normal healthy individual and to promote chronic degenerative diseases.102
  68. I.V.s (intravenous feedings) of sugar water can cut off oxygen to your brain.103
  69. Sugar increases your risk of polio.104
  70. High sugar intake can cause epileptic seizures.105
  71. Sugar causes high blood pressure in obese people.106
  72. In intensive care units: Limiting sugar saves lives.107
  73. Sugar may induce cell death.108
  74. In juvenile rehabilitation camps, when children were put on a low sugar diet, there was a 44 percent drop in antisocial behavior.109
  75. Sugar dehydrates newborns.110
  76. Sugar can cause gum disease.111

References

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  2. Ringsdorf, W., Cheraskin, E. and Ramsay R. Sucrose, Neutrophilic Phagocytosis and Resistance to Disease, Dental Survey. 1976;52(12):46_48.
  3. Couzy, F., et al. “Nutritional Implications of the Interaction Minerals,” Progressive Food and Nutrition Science 17;1933:65-87
  4. Kozlovsky, A., et al. Effects of Diets High in Simple Sugars on Urinary Chromium Losses. Metabolism. June 1986;35:515_518.
  5. Fields, M.., et al. Effect of Copper Deficiency on Metabolism and Mortality in Rats Fed Sucrose or Starch Diets, Journal of Clinical Nutrition. 1983;113:1335_1345.
  6. Lemann, J. Evidence that Glucose Ingestion Inhibits Net Renal Tubular Reabsorption of Calcium and Magnesium. Journal of Clinical Nutrition. 1976 ;70:236_245.
  7. Goldman, J., et al. Behavioral Effects of Sucrose on Preschool Children. Journal of Abnormal Child Psychology.1986;14(4):565_577.
  8. Jones, T. W., et al. Enhanced Adrenomedullary Response and Increased Susceptibility to Neuroglygopenia: Mechanisms Underlying the Adverse Effect of Sugar Ingestion in Children. Journal of Pediatrics. Feb 1995;126:171-7.
  9. Scanto, S. and Yudkin, J. The Effect of Dietary Sucrose on Blood Lipids, Serum Insulin, Platelet Adhesiveness and Body Weight in Human Volunteers, Postgraduate Medicine Journal. 1969;45:602_607.
  10. Albrink, M. and Ullrich I. H. Interaction of Dietary Sucrose and Fiber on Serum Lipids in Healthy Young Men Fed High Carbohydrate Diets. American Journal of Clinical Nutrition. 1986;43:419-428. Pamplona, R., et al. Mechanisms of Glycation in Atherogenesis. Med Hypotheses. Mar 1993;40(3):174-81.
  11. Reiser, S. Effects of Dietary Sugars on Metabolic Risk Factors Associated with Heart Disease. Nutritional Health. 1985;203_216.
  12. Lewis, G. F. and Steiner, G. Acute Effects of Insulin in the Control of Vldl Production in Humans. Implications for The insulin-resistant State. Diabetes Care. 1996 Apr;19(4):390-3 R. Pamplona, M. .J., et al. Mechanisms of Glycation in Atherogenesis. Medical Hypotheses. 1990;40:174-181.
  13. Cerami, A., Vlassara, H., and Brownlee, M. “Glucose and Aging.” Scientific American. May 1987:90. Lee, A. T. and Cerami, A. The Role of Glycation in Aging. Annals of the New York Academy of Science; 663:63-67.
  14. Takahashi, E., Tohoku University School of Medicine, Wholistic Health Digest. October 1982:41:00
  15. Quillin, Patrick, Cancer’s Sweet Tooth, Nutrition Science News. Ap 2000 Rothkopf, M.. Nutrition. July/Aug 1990;6(4).
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  17. Moerman, C. J., et al. Dietary Sugar Intake in the Etiology of Biliary Tract Cancer. International Journal of Epidemiology. Ap 1993.2(2):207-214.
  18. The Edell Health Letter. Sept 1991;7:1.
  19. De Stefani, E.”Dietary Sugar and Lung Cancer: a Case control Study in Uruguay.” Nutrition and Cancer. 1998;31(2):132_7.
  20. Cornee, J., et al. A Case-control Study of Gastric Cancer and Nutritional Factors in Marseille, France. European Journal of Epidemiology 11 (1995):55-65.
  21. Kelsay, J., et al. Diets High in Glucose or Sucrose and Young Women. American Journal of Clinical Nutrition. 1974;27:926_936. Thomas, B. J., et al. Relation of Habitual Diet to Fasting Plasma Insulin Concentration and the Insulin Response to Oral Glucose, Human Nutrition Clinical Nutrition. 1983; 36C(1):49_51.
  22. Dufty, William. Sugar Blues. (New York:Warner Books, 1975).
  23. Acta Ophthalmologica Scandinavica. Mar 2002;48;25. Taub, H. Ed. Sugar Weakens Eyesight, VM NEWSLETTER;May 1986:06:00
  24. Dufty.
  25. Yudkin, J. Sweet and Dangerous.(New York:Bantam Books,1974) 129
  26. Cornee, J., et al. A Case-control Study of Gastric Cancer and Nutritional Factors in Marseille, France, European Journal of Epidemiology. 1995;11
  27. Persson P. G., Ahlbom, A., and Hellers, G. Epidemiology. 1992;3:47-52.
  28. Jones, T. W., et al. Enhanced Adrenomedullary Response and Increased Susceptibility to Neuroglygopenia: Mechanisms Underlying the Adverse Effect of Sugar Ingestion in Children. Journal of Pediatrics. Feb 1995;126:171-7.
  29. Lee, A. T.and Cerami A. The Role of Glycation in Aging. Annals of the New York Academy of Science.1992;663:63-70.
  30. Abrahamson, E. and Peget, A. Body, Mind and Sugar. (New York: Avon, 1977.}
  31. Glinsmann, W., Irausquin, H., and Youngmee, K. Evaluation of Health Aspects of Sugar Contained in Carbohydrate Sweeteners. F. D. A. Report of Sugars Task Force. 1986:39:00 Makinen K.K.,et al. A Descriptive Report of the Effects of a 16_month Xylitol Chewing_gum Programme Subsequent to a 40_month Sucrose Gum Programme. Caries Research. 1998; 32(2)107_12.
  32. Glinsmann, W., Irausquin, H., and K. Youngmee. Evaluation of Health Aspects of Sugar Contained in Carbohydrate Sweeteners. F. D. A. Report of Sugars Task Force.1986;39:36_38.
  33. Appleton, N. New York: Healthy Bones. Avery Penguin Putnam:1989.
  34. Keen, H., et al. Nutrient Intake, Adiposity, and Diabetes. British Medical Journal. 1989; 1:00 655_658
  35. Darlington, L., Ramsey, N. W. and Mansfield, J. R. Placebo Controlled, Blind Study of Dietary Manipulation Therapy in Rheumatoid Arthritis, Lancet. Feb 1986;8475(1):236_238.
  36. Powers, L. Sensitivity: You React to What You Eat. Los Angeles Times. (Feb. 12, 1985). Cheng, J., et al. Preliminary Clinical Study on the Correlation Between Allergic Rhinitis and Food Factors. Lin Chuang Er Bi Yan Hou Ke Za Zhi Aug 2002;16(8):393-396.
  37. Erlander, S. The Cause and Cure of Multiple Sclerosis, The Disease to End Disease.” Mar 3, 1979;1(3):59_63.
  38. Crook, W. J. The Yeast Connection. (TN:Professional Books, 1984).
  39. Heaton, K. The Sweet Road to Gallstones. British Medical Journal. Apr 14, 1984; 288:00:00 1103_1104. Misciagna, G., et al. American Journal of Clinical Nutrition. 1999;69:120-126.
  40. Cleave, T. The Saccharine Disease. (New Canaan, CT: Keats Publishing, 1974).
  41. Ibid.
  42. Cleave, T. and Campbell, G. (Bristol, England:Diabetes, Coronary Thrombosis and the Saccharine Disease: John Wright and Sons, 1960).
  43. Behall, K. Influ ence of Estrogen Content of Oral Contraceptives and Consumption of Sucrose on Blood Parameters. Disease Abstracts International. 1982;431437.
  44. Tjäderhane, L. and Larmas, M. A High Sucrose Diet Decreases the Mechanical Strength of Bones in Growing Rats. Journal of Nutrition. 1998:128:1807_1810.
  45. Beck, Nielsen H., Pedersen O., and Schwartz S. Effects of Diet on the Cellular Insulin Binding and the Insulin Sensitivity in Young Healthy Subjects. Diabetes. 1978;15:289_296 .
  46. Sucrose Induces Diabetes in Cat. Federal Protocol. 1974;6(97). diabetes
  47. Reiser, S., et al. Effects of Sugars on Indices on Glucose Tolerance in Humans. American Journal of Clinical Nutrition. 1986;43:151-159.
  48. Journal of Clinical Endocrinology and Metabolism. Aug 2000
  49. Hodges, R., and Rebello, T. Carbohydrates and Blood Pressure. Annals of Internal Medicine. 1983:98:838_841.
  50. Behar, D., et al. Sugar Challenge Testing with Children Considered Behaviorally Sugar Reactive. Nutritional Behavior. 1984;1:277_288.
  51. Furth, A. and Harding, J. Why Sugar Is Bad For You. New Scientist. Sep 23, 1989;44.
  52. Simmons, J. Is The Sand of Time Sugar? LONGEVITY. June 1990:00:00 49_53.
  53. Appleton, N. New York: LICK THE SUGAR HABIT. Avery Penguin Putnam:1988. allergies
  54. Cleave, T. The Saccharine Disease: (New Canaan Ct: Keats Publishing, Inc., 1974).131.
  55. Ibid. 132
  56. Pamplona, R., et al. Mechanisms of Glycation in Atherogenesis. Medical Hypotheses . 1990:00:00 174_181.
  57. Vaccaro O., Ruth, K. J. and Stamler J. Relationship of Postload Plasma Glucose to Mortality with 19 yr Follow up. Diabetes Care. Oct 15,1992;10:328_334. Tominaga, M., et al, Impaired Glucose Tolerance Is a Risk Factor for Cardiovascular Disease, but Not Fasting Glucose. Diabetes Care. 1999:2(6):920-924.
  58. Lee, A. T. and Cerami, A. Modifications of Proteins and Nucleic Acids by Reducing Sugars: Possible Role in Aging. Handbook of the Biology of Aging. (New York: Academic Press, 1990.).
  59. Monnier, V. M. Nonenzymatic Glycosylation, the Maillard Reaction and the Aging Process. Journal of Gerontology 1990:45(4):105_110.
  60. Cerami, A., Vlassara, H., and Brownlee, M. Glucose and Aging. Scientific American. May 1987:00:00 90
  61. Dyer, D. G., et al. Accumulation of Maillard Reaction Products in Skin Collagen in Diabetes and Aging. Journal of Clinical Investigation. 1993:93(6):421_22.
  62. Veromann, S.et al.”Dietary Sugar and Salt Represent Real Risk Factors for Cataract Development.” Ophthalmologica. 2003 Jul-Aug;217(4):302-307.
  63. Goulart, F. S. Are You Sugar Smart? American Fitness. March_April 1991:00:00 34_38. Milwakuee, WI
  64. Monnier, V. M. Nonenzymatic Glycosylation, the Maillard Reaction and the Aging Process. Journal of Gerontology. 1990:45(4):105_110.
  65. Ceriello, A. Oxidative Stress and Glycemic Regulation. Metabolism. Feb 2000;49(2 Suppl 1):27-29.
  66. Appleton, Nancy. New York; Lick the Sugar Habit. Avery Penguin Putnam, 1988 enzymes
  67. Hellenbrand, W. Diet and Parkinson’s Disease. A Possible Role for the Past Intake of Specific Nutrients. Results from a Self-administered Food-frequency Questionnaire in a Case-control Study. Neurology. Sep 1996;47(3):644-650.
  68. Goulart, F. S. Are You Sugar Smart? American Fitness. March_April 1991:00:00 34_38.
  69. Ibid.
  70. Yudkin, J., Kang, S. and Bruckdorfer, K. Effects of High Dietary Sugar. British Journal of Medicine. Nov 22, 1980;1396.
  71. Blacklock, N. J., Sucrose and Idiopathic Renal Stone. Nutrition and Health. 1987;5(1-2):9- Curhan, G., et al. Beverage Use and Risk for Kidney Stones in Women. Annals of Internal Medicine. 1998:28:534-340.
  72. Goulart, F. S. Are You Sugar Smart? American Fitness. March_April 1991:00:00 34_38. Milwakuee, WI,:
  73. Ibid. fluid retention
  74. Ibid. bowel movement
  75. Ibid. compromise the lining of the capillaries
  76. Nash, J. Health Contenders. Essence. Jan 1992; 23:00 79_81.
  77. Grand, E. Food Allergies and Migraine.Lancet. 1979:1:955_959.
  78. Schauss, A. Diet, Crime and Delinquency. (Berkley Ca; Parker House, 1981.)
  79. Molteni, R, et al. A High-fat, Refined Sugar Diet Reduces Hippocampal Brain-derived Neurotrophic Factor, Neuronal Plasticity, and Learning. NeuroScience. 2002;112(4):803-814.
  80. Christensen, L. The Role of Caffeine and Sugar in Depression. Nutrition Report. Mar 1991;9(3):17-24.
  81. Ibid,44
  82. Yudkin, J. Sweet and Dangerous.(New York:Bantam Books,1974) 129
  83. Frey, J. Is There Sugar in the Alzheimer’s Disease? Annales De Biologie Clinique. 2001; 59 (3):253-257.
  84. Yudkin, J. Metabolic Changes Induced by Sugar in Relation to Coronary Heart Disease and Diabetes. Nutrition and Health. 1987;5(1-2):5-8.
  85. Yudkin, J and Eisa, O. Dietary Sucrose and Oestradiol Concentration in Young Men. Annals of Nutrition and Metabolism. 1988:32(2):53-55.
  86. The Edell Health Letter. Sept 1991;7:1.
  87. Gardner, L. and Reiser, S. Effects of Dietary Carbohydrate on Fasting Levels of Human Growth Hormone and Cortisol. Proceedings of the Society for Experimental Biology and Medicine. 1982;169:36_40.
  88. Journal of Advanced Medicine. 1994;7(1):51-58.
  89. Ceriello, A. Oxidative Stress and Glycemic Regulation. Metabolism. Feb 2000;49(2 Suppl 1):27-29.
  90. Postgraduate Medicine.Sept 1969:45:602-07.
  91. Lenders, C. M. Gestational Age and Infant Size at Birth Are Associated with Dietary Intake among Pregnant Adolescents. Journal of Nutrition. Jun 1997;1113- 1117
  92. Ibid.
  93. Sugar, White Flour Withdrawal Produces Chemical Response. The Addiction Letter. Jul 1992:04:00 Colantuoni, C., et al. Evidence That Intermittent, Excessive Sugar Intake Causes Endogenous Opioid Dependence. Obes Res. Jun 2002 ;10(6):478-488. Annual Meeting of the American Psychological Society, Toronto, June 17, 2001 www.mercola.com/2001/jun/30/sugar.htm
  94. Ibid.
  95. Sunehag, A. L., et al. Gluconeogenesis in Very Low Birth Weight Infants Receiving Total Parenteral Nutrition Diabetes. 1999 ;48 7991_800.
  96. Christensen L., et al. Impact of A Dietary Change on Emotional Distress. Journal of Abnormal Psychology.1985;94(4):565_79.
  97. Nutrition Health Review. Fall 85 changes sugar into fat faster than fat
  98. Ludwig, D. S., et al. High Glycemic Index Foods, Overeating and Obesity. Pediatrics. March 1999;103(3):26-32.
  99. Pediatrics Research. 1995;38(4):539-542. Berdonces, J. L. Attention Deficit and Infantile Hyperactivity. Rev Enferm. Jan 2001;4(1)11-4
  100. Blacklock, N. J. Sucrose and Idiopathic Renal Stone. Nutrition Health. 1987;5(1 & 2):9-
  101. Lechin, F., et al. Effects of an Oral Glucose Load on Plasma Neurotransmitters in Humans. Neurophychobiology. 1992;26(1-2):4-11.
  102. Fields, M. Journal of the American College of Nutrition. Aug 1998;17(4):317_321.
  103. Arieff, A. I. Veterans Administration Medical Center in San Francisco. San Jose Mercury; June 12/86. IVs of sugar water can cut off oxygen to the brain.
  104. Sandler, Benjamin P. Diet Prevents Polio. Milwakuee, WI,:The Lee Foundation for for Nutritional Research, 1951
  105. Murphy, Patricia. The Role of Sugar in Epileptic Seizures. Townsend Letter for Doctors and Patients. May, 2001 Murphy Is Editor of Epilepsy Wellness Newsletter, 1462 West 5th Ave., Eugene, Oregon 97402
  106. Stern, N. & Tuck, M. Pathogenesis of Hypertension in Diabetes Mellitus. Diabetes Mellitus, a Fundamental and Clinical Test. 2nd Edition, (PhiladelphiA; A:Lippincott Williams & Wilkins, 2000)943-957.
  107. Christansen, D. Critical Care: Sugar Limit Saves Lives. Science News. June 30, 2001; 159:404.
  108. Donnini, D. et al. Glucose May Induce Cell Death through a Free Radical-mediated Mechanism.Biochem Biohhys Res Commun. Feb 15, 1996:219(2):412-417.
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